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Dihexa was also predicted to be 22.59% unbound to plasma proteins in circulation, thus making it available for distribution into the tissues
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Its being studied for GERD because preclinical research shows it accelerates mucosal healing in the stomach and esophagus by upregulating VEGF and FGF, promotes angiogenesis at injury sites, and modulates inflammatory pathways implicated in reflux esophagitis

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