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showed that Baohuoside I induces apoptosis in NSCLC by activating the ROS/p38MAPK pathway, disrupting mitochondrial membrane potential, and triggering caspase activation (Song et al
Investigations on the level of H 2 S in the vitreous and plasma of PDR patients have revealed a much higher expression, indicating the potential effects of H 2 S in the pathogenesis of PDR 111
2022;132(1):e151685
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The authors thank Prof Liwei An for manuscript editing and language polishing
[DOI] [PubMed] [Google Scholar] Murillo Ortiz B
