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Keywords: BPC 157, Injury, Rats, Spinal cord Introduction We focused on the application of the stable gastric pentadecapeptide BPC 157 [111] to improve the outcomes of spinal cord injury in rats

A single autophosphorylation site on KDR/Flk-1 is essential for VEGF-A-dependent activation of PLC-gamma and DNA synthesis in vascular endothelial cells

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When a spinal segment is immobilized, there is increased load on the adjacent levels disc and facet joint which in turn cause abnormal motion and hence a resultant compensatory arthritis formation response which leads to lumbar stenosis (Fig 2)

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