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glutathione and colon cancer DLAT sustains redox homeostasis prevent colorectal from ferroptosis by regulating SLC25A39-mediated mitochondrial transport Glutathione and UDP-HexNAc as marker

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glutathione and colon cancer DLAT sustains redox homeostasis prevent colorectal from ferroptosis by regulating SLC25A39-mediated mitochondrial transport Glutathione and UDP-HexNAc as marker

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glutathione and colon cancer DLAT sustains redox homeostasis prevent colorectal from ferroptosis by regulating SLC25A39-mediated mitochondrial transport Glutathione and UDP-HexNAc as marker

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glutathione and colon cancer DLAT sustains redox homeostasis prevent colorectal from ferroptosis by regulating SLC25A39-mediated mitochondrial transport Glutathione and UDP-HexNAc as marker

Corna, G

glutathione and colon cancer DLAT sustains redox homeostasis prevent colorectal from ferroptosis by regulating SLC25A39-mediated mitochondrial transport Glutathione and UDP-HexNAc as marker

[DOI] [PubMed] [Google Scholar] 85.Cossart P., Helenius A

glutathione and colon cancer DLAT sustains redox homeostasis prevent colorectal from ferroptosis by regulating SLC25A39-mediated mitochondrial transport Glutathione and UDP-HexNAc as marker

D.HasipekM.GrabowskiD.LindnerD.PhillipsJ

glutathione and colon cancer DLAT sustains redox homeostasis prevent colorectal from ferroptosis by regulating SLC25A39-mediated mitochondrial transport Glutathione and UDP-HexNAc as marker

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