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There is a previous report that methylglyoxal interferes with GSH synthesis and secretion [18], which could be why methylglyoxal treated cells show a lower level of GSH, compared to control and CoCl 2 treated cells

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An estrogenic regulation of DAO activity has been reported in different animal models [28,29]

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TB, again the most potent of the tested compounds, significantly attenuated ROS production induced by glutamate with an EC50 of 0.11 nM (Figure 4C, Table 1)