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This is due to impairment in GSH synthetic capacity (40% lower) and not precursor limitation (cysteine levels were unchanged)

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35 The exact significance of both the former and the latter in therapeutic terms in the management of hyperpigmentation is unknown except what can be gathered from observational studies

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Selenoproteins appear to limit tumor development during the initiation phase by protecting cells from oxidative DNA damage, but they may instead promote the growth of tumor cells that have already formed and lower the likelihood that patients will survive