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This neuroinflammatory response not only contributes to disease progression, but also amplifies damage initiated by other pathological processes, such as protein aggregation, mitochondrial dysfunction, and synaptic failure

These mechanisms are investigated in cell-culture and animal models, not in humans

Nitrated lipoproteins, and cardiovascular dysfunction in patients with type 2 diabetes: what do we know and what remains to be explained

The most commonly acknowledged serotonin presentation can be from the enterochromaffin cells out of tissues into the blood, secreted luminally and basolaterally increased serotonin uptake by circulating platelets and activation after stimulation, toward the increased stimulation of myenteric and gastrointestinal motility [187]

Understanding these dimensions clarifies why urges appear and how to respond

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