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In this context, we also demonstrated that the disruption of the cellular redox buffer, controlled by GSH, increases not only the oxidative stress (Filomeni et al., 2005a), but also the endogenous physiological flux of NO in neuronal cells ( S -nitrosylation, and DNA strand breaks in neuronal cells

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[DOI] [PubMed] [Google Scholar] 105.Peier a, Kosinski j, Cox-york k, et al

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