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However, the process of creating energy also creates oxidative waste
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Coggan et al

H 2 S has been indicated as a regulator of tumor progression and metastasis in recent years [138], therefore, the disruption of sulfur metabolism through TST mutations can alter cellular redox states, leading to oxidative stress and changes in H 2 S levels

Renal tubular toxicity of acetaminophen with the development of potassium and phosphate wasting and acute tissue injury may also be linked to energy (ATP) depletion partially generated by the ATP-depleting 5-oxoproline cycle

Glucose 6-phosphate dehydrogenase from human erythrocytes: I
