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Despite strong preclinical evidence, clinical translation remains limited due to metabolic heterogeneity, adaptive rewiring, systemic toxicity, and the lack of robust predictive biomarkers

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doi: 10.1177/09645284211056663 95 TeedeHTassoneECPiltonenTMalhotraJMolBWPenaAet al

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Interestingly, extracellular 13-HODE was reported to induce mitochondrial dysfunction and airway epithelial apoptosis, which was associated with its interaction with the transient receptor potential cation channel, vanilloid type 1 (TRPV1) receptor 59