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Neuroprotective effects of acetyl-l-carnitine on lipopolysaccharide-induced neuroinflammation in mice: Involvement of brain-derived neurotrophic factor

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However, the programmed disorders at later life were not effective with supplementation of B12 but reversed through suppression of SBP- 1/SREBP1-lipogenesis signaling

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Compared with active comparators, ALC was associated with a more favorable adverse event profile, further supporting its potential role as a safer therapeutic option, especially for vulnerable populations
