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In hypoxia, the increased expression of the mitochondrial gene might involve the phosphorylation of the AKT signaling pathway, rather than the activation of ERK that is activated in a traumatic neuronal injury through PKC (77)

However, this particular dose is indicated for sleep-related pathologies such as jet-lag and works by targeting the central nervous system where canonical melatonin receptors are abundantly expressed (69)

Key pathways such as c-MET and TGF-, modulated by factors like MACC1, are central to this process

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[DOI] [PMC free article] [PubMed] [Google Scholar] 7.Chen K., Zhang J., Beeraka N.M., Tang C., Babayeva Y.V., Sinelnikov M.Y., Zhang X., Zhang J., Liu J., Reshetov I.V., et al

2018), which reflects a possible antiapoptotic potential for astaxanthin