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While the specific origins of atherosclerosis are unknown, most ideas about its onset and development concentrate around interrupting normal regulatory systems, resulting in oxidative stress

Specifically, intra-articular injection or systemic administration of specific ferroptosis inhibitors such as ferrostatin-1, or pharmacologically for example, using natural products or genetically for example, using overexpression vectors upregulating GPX4 expression, activating SLC7A11, or the key transcription factor Nrf2, can, to varying degrees, restore cartilage morphology, reduce chondrocyte death, and significantly lower the expression levels of ECM degradation markers in OA animal models

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mutation of the gene is associated with methylmalonic aciduria and homocystinuria cblc type