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These results indicate that the mitochondrial function modifier L-carnitine provides a simple and novel therapeutic strategy for NASH

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The secondary endpoint was alleviation of hepatic encephalopathy and improved results in the NCT-B

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Type 2: It is associated with chronic anxiety and displays symptoms throughout the day

In particular, ACh enhanced B cell differentiation into IgG-producing plasma cells by increasing lipid influx via CD36 and boosting mitochondrial respiration and fatty acid oxidation, leading to an autoreactive phenotype (195)