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doi: 10.3390/cells11020307

M8005) for click-chemistry investigation

Analysis of metabolically compromised tissues reveals pronounced activation of these UPR components, particularly within AT, where UPR activation is significantly increased, with insulin upregulating the UPR dose-dependently over the entire physiological insulin range (from approximately 35 to 1,450 pmol/L) (97)

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However, three pathways independent of GPX4 have been identified in the past few years, including ferroptosis suppressor protein 1 (FSP1)/coenzyme Q10 (CoQ10), GTP cyclohydrolase 1 (GCH1) and dihydroorotate dehydrogenase (DHODH)