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New insights on the mechanisms of valproate-induced hyperammonemia: inhibition of hepatic N-acetylglutamate synthase activity by valproyl-CoA
10.3389/fphys.2017.00848 Front Physiol
A study by 31P and 1H NMR spectroscopy
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Moreno-Justicia, R
Therefore, FAs cannot be accumulated as an energy source (71), leading to increased ROS production (72)
