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Ferroptosis has an iron-dependent accumulation of reactive oxygen species, depleting glutathione and causing lipid peroxidation and progression of ferroptosis [20]

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Beyond calcium signaling, BDNFTrkB-T1 interactions also regulate astrocytic function by modulating the activity of the GABA transporter GAT-1, thereby influencing GABA uptake dynamics [352]

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Empirical Valence Bond Simulations Suggest a Direct Hydride Transfer Mechanism for Human Diamine Oxidase
