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IGFBP-2 was shown to be specifically involved in endothelial recruitment, angiogenesis, and metastatic colonization, an effect that required the presence of both IGF-1 and IGF1R [40]

44,45 To date, out of these gene therapy clinical trials, only the vector with the gene encoding for increased production of glutamic acid decarboxylase has demonstrated beneficial motor function

This is a mechanistic inference, not a human finding, but it illustrates why unregulated IGF-1R agonism is not treated casually in the scientific literature