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Using a model of induced DSBs 28 , we found increased DSBs and delayed repair following suppression of KG using the IDH1 inhibitor, which was rescued by KG, l-carnitine and acetylcarnitine (Fig

Sem sua funo, os corpos gordurosos se acumulariam nos tecidos musculares e adiposos, bem como nas artrias

Dan Med Bull 1984, 31: 121-145

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Foitzik, K., Hoting, E., Heinrich, U., Tronnier, H., and Paus, R

The inability to adapt to the different cellular and environmental needs/stressors results in metabolic inflexibility, which ultimately leads to disease states like insulin resistance, lipid accumulation, and an inability to maintain homeostatic balance [24,25,26] (Figure 6)