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These findings collectively suggest a plausible mechanism whereby GSDMD-N could be transferred indirectlypotentially via extracellular vesiclesfrom supporting cells to hair cell membrane and directly inducing pyroptosis

These findings demonstrate that compounds activity partially depends on TrkB kinase activation and could suggest also non-TrkB mediated effects of GSB-106
Metabolic reprogramming in macrophages and dendritic cells in innate immunity

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Always reconstitute and inject separately, even if administering both on the same day

doi:10.1016/j.jep.2025.119705 111
