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The unexpected activity can be explained by the formation of variable amounts of disulfide-bridged PfGrx C32S/C88S homodimers and potential Diamide-modified monomers, summarized as PfGrx C32S/C88S (SX), as revealed by non-reducing SDS-PAGE (Fig

[DOI] [PubMed] [Google Scholar] 72.Cheung W, Keski-Rahkonen P, Assi N, Ferrari P, Freisling H, Rinaldi S, Slimani N, Zamora-Ros R, Rundle M, Frost G, et al

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Its ability to target multiple cellular mechanisms responsible for cancer progression and survival offers a promising avenue for developing more effective cancer treatments

Thurston RC, Kubzansky LD, Kawachi I, Berkman LF

Iron overload promotes M1 macrophage polarization and increases the M1 macrophage markers levels (IL-6, IL-1, and TNF-), leading to synovial damage