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This can trigger mitochondrial dysfunction and the production of additional ROS.Mitochondrial Activity: Neurons are highly dependent on mitochondria for energy production, and mitochondrial respiration is a significant source of ROS

Moreover, in neurological and hepatic contexts, butylphthalide mitigates traumatic brain injury-induced ferroptosis by inhibiting the AhR-CYP1B1-ROS pathway (53), while CYP1B1 drives ferroptosis in hepatocellular and cholangiocarcinoma (41)

Multi-modality treatment of primary cardiac sarcomas

Elsevier: 195213

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