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Mitochondrial dysfunction and oxidative stress in metabolic disordersA step towards mitochondria based therapeutic strategies

Overexpression of PHB2 can activate mitophagy, restore mitochondrial activity, enhance myocardial cell viability, and inhibit endotoxin-induced mitochondrial dysfunction in myocardial cells [15]

This changing course is one of the reasons lupus can be difficult to control

These modifications contribute to psoriasis pathogenesis by altering intercellular signaling, immune responses, and keratinocyte proliferation [38]

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Side effects are generally mild and temporarymostly headache, brief dizziness, or flushing