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Description
In brain tissue, misfolded proteins such as amyloid-beta and tau can sequester copper ions, creating local copper deficiency while simultaneously generating reactive oxygen species through aberrant metal-protein interactions
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Senolytic FOXO4-DRI selectively eliminates senescent fibroblasts by inducing nuclear exclusion of p53 phosphorylation, highlighting the role of the senescent microenvironment in keloid overgrowth and relapse.
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