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Indeed, it was suggested that DiY cross-linking inhibits primary nucleation and subsequently retards fibril elongation and fibrilfibril interactions (OMalley et al., 2014)
Furthermore, Lewy bodies and abnormal aggregates of the protein alpha-synuclein contribute to neuronal dysfunction and the clinical symptoms of the disease
Future clinical trials specifically designed to evaluate this combination will be essential in determining its safety profile and optimal use
single evaluation
Malavasi F, Deaglio S, Damle R, Cutrona G, Ferrarini M, Chiorazzi N
Islam Z, Pestka JJ
