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JAMs, claudin, occludin, and tricellulin have been extensively studied and shown to be involved in TJ permeability in intestinal epithelial cells

In addition, FOXO4 inhibition in cells that were already senescent, but not their control counterparts, reduced viability and cell density (Fig

Upon the release of zonulin into the lumen as a result of intestinal exposure to gliadin, zonulin can interact with two receptors, epidermal growth factor receptor (EGFR) and protease-activated receptor 2 (PAR2), which results in increased intestinal permeability (27, 32)

Additionally, while both groups presented more platform location crosses than the control group did, this difference did not reach statistical significance (Fig

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