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Besides HSRs, inflammation has been associated also with paclitaxel-induced CIPN [7], and IL-8, in particular, emerged as a crucial factor in paclitaxel-induced neuronal toxicity, which can be reduced by the inhibition of IL-8 receptors CXCR1 and CXCR2 [10, 11]

Benlian P, De Gennes JL, Foubert L, Zhang H, Gagne SE, Hayden M

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Inhibition of glucose-6-phosphate dehydrogenase activity attenuates right ventricle pressure and hypertrophy elicited by VEGFR inhibitor + hypoxia

References Garcia JM, Merriam GR, Kargi AY

doi: 10.3390/antiox12091734 61 SanhuezaSSimnLCifuentesMQuestAFG