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On the other hand inactivation of the 20S proteasome by UVA in dermal fibroblasts leads to an activation of activator protein-1 (Ap-1), controlling MMP-1 (matrixmetalloprotease-1) expression [220]

Park et al., 2022

Sevier C.S., Qu H., Heldman N., Gross E., Fass D., and Kaiser C.A

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identified NR1D1 as a potential antimicrobial therapeutic target because it promotes autophagy and lysosomal biogenesis through positive regulation of TFEB expression