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The potential immune mechanisms of UC include impacting the innate and adaptive immune response by the influence of the DCs function, dysregulation of the Treg/Th cells, promotion of pro-inflammatory factor secretion, reduction of anti-inflammatory factor secretion

This fact is justified by the toxic character of the persistent excess of glucose in the organism that ends up promoting glycation of proteins, hyperosmolarity and increase in the levels of sorbitol inside the cells [35]

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