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Membrane damage by PFPs induces the leakage of Tb 3+ into the external buffer containing DPA, leading to a fluorogenic reaction between Tb 3+ and DPA and the formation of the Tb 3+ -DPA complex, which is 10,000 times more fluorescent than the free Tb 3 [193]

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Experimental findings indicate a predominant activity within pulmonary tissue, with secondary involvement observed in the gastrointestinal tract

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