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The mode of cell death induced by these compounds was surprisingly found to be non-apoptotic, as cells treated with erastin and RSL3 died in the absence of apoptotic hallmarks [11, 12, 13], and in cells where the core apoptosis machinery caspases, BAX, and BAK was suppressed [14]

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These 18 oxidative stress markers included glutathione metabolism, transmethylation cycle and transsulfuration pathways, vitamins, and trace elements

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In dermatology, NO is implicated in mechanisms underlying inflammatory or immune-mediated dermatoses, skin infections, skin cancers, and wound healing (118)

Reference: Krivic A, Majerovic M, Jelic I, Seiwerth S, Sikiric P