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Kawamata, Y

Abstract Despite strong evidence for NMDA receptor (NMDAR) hypofunction as an underlying factor for cognitive disorders, the precise roles of various NMDAR subtypes remains unknown

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Electrophysiological studies in cortical and hippocampal neurons have shown that exogenous glycine or D-serine can further potentiate NMDA receptormediated currents, indicating that endogenous co-agonist concentrations remain below saturating levels at least in a subset of synapses (Wilcox et al

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