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As part of its wound healing effect, GHK-Cu has been shown to contribute to the initial upregulation of growth factors and inflammatory mediators during nerve regeneration [8]

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Further supporting agonist-induced large-scale LBD rearrangements, 3-D variability analysis (3DVA) of the intact GluK2/GluK5 cryo-EM map revealed a rocking motion of the LBDs away from the midline, resulting in a decrease in compaction along the inter-dimer A-B and C-D interfaces (Fig

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