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-Synuclein and tau concentrations in cerebrospinal fluid of patients presenting with parkinsonism: a cohort study

This mechanism requires specific receptor expression and peptide binding capability

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Crucially, in all three cell lines, the de novo GSH synthesis inhibitor buthionine sulfoximine (BSO) abolished the ability of MPA and Pyr pretreatment to inhibit ferroptosis, consistent with a GSH-dependent protective mechanism (Fig 4B)

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