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Its complex pathophysiology shares significant mechanistic parallels with neurodegenerative processes, including neuroinflammation, synaptic dysfunction, excitotoxicity, and aberrant neural network reorganization

doi: 10.1016/s0024-3205(00)01001-8 73 SkinnerM

Bannai S, Tsukeda H, Okumura H

doi: 10.3390/metabo12090826

We believe that our novel approach targeting the defective inhibition of IL-2R second messenger pathways in Tregs from patients with autoimmune diseases represents a highly innovative and promising strategy for potentially treating a wide range of autoimmune diseases

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