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Inhibitors and antibody fragments as potential anti-inflammatory therapeutics targeting neutrophil proteinase 3 in human disease

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Hiroshige T, et al

Discussion Among non-HLA antigens reported to be involved in Tx damage ( In addition to non-HLA Abs induced by an alloimmune mechanism, Abs directed to self-antigens, such as angiotensin II type 1 receptor (AT1R), perlecan, endothelin type A receptor (ETAR), vimentin, or Rho GDP-dissociation inhibitor 2 (ARHGDIB), have been found to be associated with inferior graft outcome, although their causal role in the pathogenesis of damage is unclear ( de novo GSTT1 Ab pool