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Several pathways have been shown to be involved in the regulation of mitophagy, but the precise underlying molecular mechanisms remain elusive (Fig

[2] [5] High doses of acetaminophen are known to deplete endogenous glutathione stores, particularly in the liver, which may increase the risk of toxicity

If they do, this would also add evidence that this NNMT inhibitor improves muscle performance through increasing NAD+ levels

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