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In histology, all kidney tissues from the mice treated with colistin showed elevated expression of the AKI markers Ngal and Kim-1, tubule dilation, loss of the brush border membranes in the proximal tubules, and the formation of protein casts throughout the kidney tissue, indicating vast tubular damage (Fig
Lever M, George PM, Elmslie JL, Atkinson W, Slow S, Molyneux SL, et al
The etiology of Peyronies disease: pathogenesis and genetic contributions
10.3109/10582452.2012.704141 [DOI] [Google Scholar] 18.Wallace DJ, Linker-Israeli M, Hallegua D, Silverman S, Silver D, Weisman MH
Determinants of colonic barrier function in inflammatory bowel disease and potential therapeutics
The Effects of the Paleo Diet on Autoimmune Thyroid Disease: A Mixed Methods Review
