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While our study focused on endothelial K-Hcy-HMGB1/2-mediated senescence, K-Hcy modifications may constitute a novel pathological axis that disrupts multiple proteins and cellular processes, thereby driving ED through broader and previously unrecognized mechanisms

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While the loss of dopaminergic neurons in the substantia nigra remains the hallmark of the disease, growing evidence highlights the significant involvement of other neurotransmitter systems, including noradrenergic, serotonergic, cholinergic, glutamatergic, and GABAergic pathways, in disease progression and symptom variability

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