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Reactive oxygen species mediate arachidonic acid-induced dilation in porcine coronary microvessels
Neurosci [Internet]
In fact, animal models of SARS have been developed to recapitulate these findings
Co-cultures of glioma and neuronal cells demonstrated that the released l-Glu was sufficient to induce excitotoxic damage and that the neuronal loss could be reduced by either by preventing its efflux through Sx c - (4- S -CPG or SSZ) or inhibiting its action at NMDA receptors (MK-801)
Obeid, R
