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Hepcidin expression in iron overload diseases is variably modulated by circulating factors

Reddy, V., Bhandari, S

Quick answer: Performance evidence is mixed

Another example of a significant genetic predisposition is a polymorphism of PTPN22 that increases RA-risk in SE carriers [37]

doi: 10.1016/j.cgh.2018.10.021 67
