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Description
R., TadinStrapps M., Mol
doi: 10.3390/endocrines5020011
Exp Mol Med 2020;52:1530
Riboflavin, in the form of flavin adenine dinucleotide (FAD), is shown as the required cofactor for this reaction, which consumes NADPH and a proton (H + ) and releases NADP +
This represents a detrimental phenomenon observed in patients with poor response to subsequent therapies
It is also unusually stable in gastric acid, which is why oral dosing is even theoretically plausible
