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With exacerbated oxidative stress and cytokine storm, increased levels of interleukin-6 (IL-6) and decreased levels of interferon- (IFN- ) are thought to contribute to worsening infection and are also associated with decreased levels of GSH [11]

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In contrast, under oxidative stress NRF2 dissociates from KEAP1 and translocates to the nucleus, where it activates the downstream expression of antioxidative response element (ARE)-containing target genes, many of which have been shown to play a role in defending against ferroptosis [65, 66]