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Myo-inositol oxygenase overexpression accentuates generation of reactive oxygen species and exacerbates cellular injury following high glucose ambience: A new mechanism relevant to the pathogenesis of diabetic nephropathy

The cellular contribution to aerobic glycolysis is yet unclear and is a matter of debate (Dienel and Cruz, 2016

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The role of this risk profile can be related to the immune response and angiogenesis, enhancing inflammatory activity and attracting immunosuppressive cell infiltration to form a chronic inflammatory microenvironment, which promotes GBM growth (37)

Shelf Res