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An increase in blood flow within muscles can boost protein synthesis, and L-citrulline can upregulate the expression of peroxisome proliferator-activated receptor- coactivator-1 (PGC-1), which is regarded as associated with neovascularization [108]

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Previous studies have suggested that macrophages, neurogenic inflammation, lipid peroxides, and pain-inducing prostaglandins play a vital role in the pathophysiology of endometriotic pain [46,91,92,93]