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doi: 10.1016/j.fertnstert.2004.02.121

L-carnitine is a key element of the last phase of fat decomposition

At least in mice, ACLY deletion 6 or bempedoic acid 40 strongly suppresses glucose-dependent hepatic de novo lipogenesis, suggesting that ACLY is a dominant mechanism for glucose-dependent de novo lipogenesis in the liver, although acetate can sustain lipogenesis in the absence of ACLY 6

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Inhibition of mitochondrial (etomoxir) and peroxisomal (thioridazine) fatty acid oxidation revealed that each organelle contributes to approximately half of acetyl-CoA produced from palmitate in the DKO cells (Supplemental Figure 5G)