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In the current review, five eligible studies were excluded from quantitative synthesis due to incomplete/non-extractable data
The approaches are designed to recognize and bind to specific mtDNA sequences, induce a double-strand break, and initiate targeted degradation of the mutant mtDNA in a heteroplasmic population
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In addition, accumulation of apoC-III-rich apoB-containing lipoproteins might have direct atherogenic consequences
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