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While ferroptosis and gut dysbiosis have been extensively studied in isolation, growing evidence supports profound molecular crosstalk between these pathways, which synergistically drive OA progression

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Recent evidence has suggested that GPx1 and c-Src tyrosine kinases participate in the phosphorylation of IB which, in response to hypoxia, leads to the activation of NF-B elevated in hydrogen peroxide-treated embryonic fibroblasts [50]

Gargano ML, van Griensven LJLD, Isikhuemhen OS, et al

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