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Damage to axons is mediated by the following: (1) failure in mitochondrial energy metabolism due to inhibition of the respiratory chain by nitric oxide, which in turn causes a decrease in Na + /K + ATPase activity and alters Na + dependent glutamate transporters, (2) overexpression of glutamate receptors, (3) oligodendroglial excitotoxicity, (4) massive influx of extracellular Ca ++ , (5) activation of proteases, and (6) impaired axonal transport

doi: 10.1016/j.aqrep.2022.101114

Georgiades M, Alampounti A, Somers J et al (2023) Hearing of malaria mosquitoes is modulated by a beta-adrenergic-like octopamine receptor which serves as insecticide target

Inhibition of interferon, cytokine, and lymphocyte proliferative responses in elite swimmers with altitude exposure

A bridging [4Fe4S] cluster and nucleotide binding are essential for function of the Cfd1Nbp35 complex as a scaffold in ironsulfur protein maturation

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