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33 Understanding the etiological contributors to such donordependent variance might shed light not just on RBC storage biology, but also contribute significantly to our understanding of those pathologies associated with RBC hemolysis due to exacerbated oxidant stress (e.g., sickle cell disease, b-thalassemia, pulmonary hypertension, Down syndrome, inflammaging 39 )

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In a phase III randomized controlled trial of 807 infants in 14 university hospitals across the United States, Matthew et al

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